Atrial fibrillation is an irregular heart rhythm that starts in the upper parts (atria) of the heart causing irregular beating between the atria and the lower parts (ventricles) of the heart. The lower parts may beat fast and without a regular rhythm. Symptoms of atrial fibrillation include dizziness, light-headedness, shortness of breath, chest pain and irregular heart beat.
In some cases, devices are attached to the catheter, which can be used to seal congenital defects that have caused a hole in the heart. By reducing the size of the hole, the device can help the heart heal the hole over time.
Imagine the energy as bright yellow going up your governing vessel and bright white going down your conception vessel. There are more sensations attached to these vessels, but I don't want to overload you right now. At the moment, imagine your energies flowing smoothly through your two main vessels. Inhale deeply to lead your "Qi" through your governing vessel. Exhale deeply to lead your "Qi" through your conception vessel; this is grand circulation.
After an investigation of the diagnostic analysis of the situation, treatment options should be taken immediately to avoid the fate of the shape of a heart attack. Usually, the diagnosis is based on the results of ultrasound and X-rays These methods are based on the results of tests designed to stress the muscle thickness, size and function of the study and heart function. The structure of the heart valves, kidney or stenosis of potential and muscle performance to be identified and evaluated before taking one or several treatment options available.
Some people may treat acid reflux and chest pains as dismissible and can easily be remedied by antacids. The problem now lies with the similarity between acid reflux – chest pains as against the chest pains that are onsets to a heart attack. Thus, there is the real need to know the underlying differences between these two conditions since the latter could prove to be fatal if ignored.
Doctors are still speculating about the mechanism that causes a plaque deposit to burst. There is, however, one theory that has more adherents than most. The theory is that, as the level of LDL increases in the blood, some of it seeps into the lining of the coronary arteries. Macrophages are alerted to the presence of an invader and enter the tissue to repel the foreign material. If the cytokine signals increase the inflammation process instead of winding it back, the plaque will become unstable. This causes the plaque deposit to burst and block a blood vessel.